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MatchTypeWhy
Bear, ChristinePerson Why?
Chloride-Bicarbonate AntiportersConcept Why?
The chloride channel ClC-4 co-localizes with cystic fibrosis transmembrane conductance regulator and may mediate chloride flux across the apical membrane of intestinal epithelia.Academic Article Why?
Amelioration of intestinal disease severity in cystic fibrosis mice is associated with improved chloride secretory capacity.Academic Article Why?
ClC-2 channels regulate neuronal excitability, not intracellular chloride levels.Academic Article Why?
ClC-2 contributes to native chloride secretion by a human intestinal cell line, Caco-2.Academic Article Why?
Increased prevalence of CFTR mutations and variants and decreased chloride secretion in primary sclerosing cholangitis.Academic Article Why?
Non-CFTR chloride channels likely contribute to secretion in the murine small intestine.Academic Article Why?
Quaternary structure of the chloride channel ClC-2.Academic Article Why?
Swelling-induced chloride current in glioblastoma proliferation, migration, and invasion.Academic Article Why?
Prescott, StevePerson Why?
Congenital chloride-losing diarrhea causing mutations in the STAS domain result in misfolding and mistrafficking of SLC26A3.Academic Article Why?
Cell volume changes and the activity of the chloride conductance path.Academic Article Why?
Chloride regulation in the pain pathway.Academic Article Why?
Extracellular chloride regulates mesangial cell calcium response to vasopressor peptides.Academic Article Why?
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